• Medientyp: E-Artikel
  • Titel: EBNA-1 specific effector T cell deletion associated with holoendemic malaria exposure in the etiology of endemic Burkitt’s lymphoma (P3063)
  • Beteiligte: Mulama, David; Chelimo, Kiprotich; Collins, Ouma; Jura, Walter; Otieno, Juliana; Vulule, John; Munz, Christian; Moormann, Ann
  • Erschienen: The American Association of Immunologists, 2013
  • Erschienen in: The Journal of Immunology
  • Sprache: Englisch
  • DOI: 10.4049/jimmunol.190.supp.187.7
  • ISSN: 0022-1767; 1550-6606
  • Schlagwörter: Immunology ; Immunology and Allergy
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  • Beschreibung: <jats:title>Abstract</jats:title> <jats:p>Etiological mechanism of eBL remain largely unknown despite long-standing epidemiological link between malaria and EBV co-infection. Age-dependent deficiency in EBV-specific CD8+ T cell-mediated IFN-γ immunosurveillance has been reported in malaria holoendemic exposed children which has been associated with eBL diagnosis. We sought to investigate whether qualitative differences in EBNA-1 specific T cells existed in malaria-exposed children to explain their increased risk for eBL. We conducted a cross-sectional study of healthy children from western Kenya with divergent malaria exposure:Kisumu (holoendemic) and Nandi (hypoendemic) and eBL age-matched patients. T cell effector function was evaluated by multiparameter flow cytometry against T cell lineage markers(CD3,4,8,45RA &amp; CCR7) and functional phenotype (IFN-γ, IL10, IL17 &amp; PD1) to EBNA-1. EBNA-1 specific responses were compared to MSP-1, EBNA2 and EBNA3A. We found significantly higher IFN-γ responses to MSP1 in the eBL &amp; Kisumu compared to Nandi children (p=0.03), in contrast to EBNA1, which were significantly lower in eBL and Kisumu compared to Nandi children (p=0.01). Responses to EBNA2 and EBNA3A did not differ between groups. IFN-γ was generated from CD4 TEM and a heterogeneous combination of CD8 T-cells. PD-1 expressing T-cells were less frequent in eBL patients and highest in Kisumu children.This suggests that the EBNA1-specific deficiencies are due to deletion of T-cell population due to malaria induced exhaustion.</jats:p>
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